Search results for "CORTEX EXCITABILITY"

showing 5 items of 5 documents

Altered processing of sensory stimuli in patients with migraine.

2014

Migraine is a cyclic disorder, in which functional and morphological brain changes fluctuate over time, culminating periodically in an attack. In the migrainous brain, temporal processing of external stimuli and sequential recruitment of neuronal networks are often dysfunctional. These changes reflect complex CNS dysfunction patterns. Assessment of multimodal evoked potentials and nociceptive reflex responses can reveal altered patterns of the brain's electrophysiological activity, thereby aiding our understanding of the pathophysiology of migraine. In this Review, we summarize the most important findings on temporal processing of evoked and reflex responses in migraine. Considering these d…

TRANSCRANIAL MAGNETIC STIMULATIONSensory processingmedicine.medical_treatmentMigraine DisordersThalamocortical dysrhythmiaEVENT-RELATED POTENTIALSINTENSITY-DEPENDENCESensory systemElectroencephalographyCellular and Molecular Neurosciencesensory stimuli migraine neurophysiology thalamo-cortical dysrtmia.Event-related potentialNociceptive ReflexPhysical StimulationPHASE SYNCHRONIZATION CHANGESReflexMedicine and Health SciencesmedicineHumansHIGH-FREQUENCY OSCILLATIONSEvoked PotentialsMigraineNOCICEPTIVE BLINK REFLEXCONTINGENT NEGATIVE-VARIATIONMEDICATION-OVERUSE HEADACHEmedicine.diagnostic_testbusiness.industryBrainElectroencephalographyAUDITORY-EVOKED-POTENTIALSmedicine.diseaseMigraineconnectivitySensation DisordersReflexVISUAL-CORTEX EXCITABILITYNeurology (clinical)businesssynchronizationNeuroscienceNature reviews. Neurology
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Modulation of visual cortex excitability in migraine with aura: effects of valproate therapy.

2009

We explored the effects of valproate treatment on visual cortex excitability changes in migraine with aura patients. Abnormal cortical excitability has been suggested to play an important role in the etiopathogenesis of migraine; in particular, it has been suggested a failure of inhibitory circuits in migraine with aura. Valproate acts as a central GABA agonist and it is reasonable suppose that VPA could modify cortical excitability state. Phosphene threshold (PT) was assessed at baseline and after 1 Hz rTMS before and after one month therapy. We found that low-frequency rTMS in drug-free migraineurs decreased PT, while the treatment with the GABA agonist valproate is able to revert the eff…

AgonistAdultMaleTime Factorsmedicine.drug_classGABA Agentsmedicine.medical_treatmentMigraine with AuraPhosphenesSettore BIO/09 - FisiologiaYoung AdultCortex (anatomy)medicineHumansvisual cortex excitability migraine with auraVisual CortexValproic AcidAnalysis of VarianceGeneral NeuroscienceValproic AcidMiddle Agedmedicine.diseaseTranscranial Magnetic StimulationMigraine with auraAnticonvulsantVisual cortexmedicine.anatomical_structurePhosphenenervous systemMigraineSettore MED/26 - NeurologiaFemalemedicine.symptomPsychologyNeurosciencemedicine.drugNeuroscience letters
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Lack of effects of low frequency repetitive transcranial magnetic stimulation on alpha rhythm phase synchronization in migraine patients

2010

The study aimed to test the modulation induced by 1 Hz repetitive Transcranial Magnetic Stimulation (rTMS) of the occipital cortex on the alpha phase synchronization under repetitive flash stimuli in 15 migraine without aura patients compared to 10 controls. The EEG was recorded by 7 channels, while flash stimuli were delivered at 9, 18, 21 and 24 Hz in basal, rTMS (15 min of 1 Hz stimulation of the occipital cortex) and sham conditions. Migraine patients displayed increased alpha-band phase synchronization under visual stimulation, while an overall desynchronizing effect was evident in controls. The rTMS resulted in a slight increase of synchronization index in migraine patients, which did…

AdultMaleMigraine Disordersmedicine.medical_treatmentStimulationElectroencephalographyVisual cortex excitabilityYoung AdultCortex (anatomy)medicineHumansCortical SynchronizationMigrainemedicine.diagnostic_testGeneral NeuroscienceBrainMiddle Agedmedicine.diseaseTranscranial Magnetic StimulationEEG synchronizationTranscranial magnetic stimulationAlpha RhythmElectrophysiologyVisual cortexmedicine.anatomical_structureMigraineFemalePsychologyNeuroscienceCortical SynchronizationNeuroscience Letters
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Motor intracortical inhibition in PD: L-DOPA modulation of high-frequency rTMS effects

2007

Dopaminergic drugs and deep brain stimulation restore cortical inhibition in Parkinson disease (PD) patients. High-frequency rTMS was also found to increase cortical inhibition in PD but its therapeutic effect is still controversial. Here we hypothesize that, if dopaminergic drugs reverse to normal cortical excitability in M1, the effect of high-frequency (hf)-rTMS in PD patients could depend on whether they are in a medicated or unmedicated state. The present study aims to explore the lasting effects of sub-threshold hf rTMS trains over M1 on cortical inhibition in patients with "on" and without "off" L-DOPA treatment. Fourteen PD patients were examined twice while "on" and "off" medicatio…

Malemedicine.medical_specialtyNeurologyDeep brain stimulationCORTICAL SILENT PERIODmedicine.medical_treatmentStimulationMotor ActivityInhibitory postsynaptic potentialPAIRED-PULSE INHIBITIONAntiparkinson AgentsLevodopaDegenerative diseaseEVOKED-POTENTIALSmedicineHumansCORTEX EXCITABILITYAgedAged 80 and overSUBTHALAMIC NUCLEUSPARKINSONS-DISEASE PATIENTSGeneral NeuroscienceDopaminergicMotor CortexNeural InhibitionParkinson DiseaseMiddle Agedmedicine.diseaseFACILITATIONCombined Modality TherapyTranscranial Magnetic StimulationTranscranial magnetic stimulationBRAIN-STIMULATIONmedicine.anatomical_structureSettore MED/26 - NeurologiaFemaleREPETITIVE TMSPsychologyNeuroscienceMotor cortexExperimental Brain Research
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Impaired glutamatergic neurotransmission in migraine with aura? Evidence by an input-output curves transcranial magnetic stimulation study.

2011

Objective: An imbalance between activity of inhibitory and facilitatory intracortical circuits could play a central role in migraine etiology. We used input-output curves to achieve further information about intracortical excitability of motor cortex in migraine with aura. METHODS: Input-output curves were measured in the right abductor pollicis brevis muscle at rest in 12 patients suffering from migraine with aura and 8 healthy subjects. Stimuli were delivered at intensity ranging from 100% to 160% of resting motor threshold with 10-second inter-stimulus intervals. Seven patients were studied before and during treatment with levetiracetam. RESULTS: Results showed a greater motor-evoked pot…

levetiracetaminput–output curvemigraine with auramagnetic stimulationmotor cortex excitability
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